Short answer first, then the reasoning. Training does two things here and only one of them is on the scale. It protects lean mass — which is the reason to do it — and it contributes modestly to the deficit, which is the reason people start and then quit when the arithmetic disappoints them. In a deep deficit, resistance work is the priority and high-volume cardio competes with recovery.
My mood has been flatter since about month four and I genuinely cannot work out whether that is the drug, the deficit, or the fact that I no longer have my main coping mechanism.
What I am after is how people separated a drug effect from the ordinary consequences of a large deficit and disrupted sleep, because I cannot.
Tell me what I have not thought of.
Dr.PeteFamMed said:Training does two things here and only one of them is on the scale.
Agreed, with the caveat that "exercise" for someone with mobility limitations is a different set of options, and the standard advice is written as though everyone can walk for an hour.
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Browse GL Biochemgreg_boulder said:My mood has been flatter since about month four and I genuinely cannot work out whether that is the drug, the deficit, or the fact that I no longer…
Same pattern here, and in the same order. Posting only so the count is not one.
Clinical perspective, offered as context rather than as advice.
Recovering from an eating disorder and using mental health: this is a complex situation and I want to be transparent about it.
I have a history of binge eating disorder. My psychiatrist, therapist, and prescribing doctor all collaborate on my care. The GLP-1 agonist has actually been therapeutic — it removes the biological urgency of binge impulses without relying on restriction.
This is NOT appropriate for all ED patients. Anorexia, bulimia, and restrictive EDs require different approaches. But for BED specifically, the evidence and my personal experience are positive. Always involve your mental health team.